• 0 Posts
  • 149 Comments
Joined 3 years ago
cake
Cake day: October 4th, 2023

help-circle





  • For internet access Plex is by far the easiest. You can use Jellyfin but it can be a lot more effort and can be brittle. Tailscale might be a solution but if you want to share with friends it would mean giving them access to your Tailscale network. Then you’ve got reverse proxies like Nginx Reverse Proxy. This would require buying a domain and configuring something like Cloudflare too, plus port forwarding on your router. Tailscale offers a publically accessible domain now which is similar but you cannot configure the TLD. Still, you’re opening an internet accessible port for a FOSS application and this is far less secure unless you know what you’re doing.


  • I think this is the last hurdle with the arr setup: discoverability. Plex has tried to jam in something, but it’s far from good. They’re never going to produce a pirate watchlist, so it would have to fall to Jellyfin. What people are seeking is the Netflix experience of “curated” content, spoonfed, and instantly watchable.

    For the record I do the same as you. I think the intentionality is a healthy barrier to mindless browsing and consumption, but once people are hooked, it’s hard to wean them off.


  • There is no evidence that lead poisoning, for example, can be inherited. There is some research that certain epigenetic changes can be inherited. The context is sometimes war and famine. But that is 2-4 degrees of abstraction away from your question, and we are many decades away from any kind of causative conclusion to that.

    If I were to steelman your position, I think there are simpler changes to point to as potential causes here. Social media. Screen time. Time spent outside and exercising. Obesity. Time spent socialising. The new “permissive” method of parenting and teaching.


  • You’re going to have to elaborate on what a genuine disability is there chief. Let me help you out:

    The UK Equality Act defines a person disabled if they have a physical or mental impairment, and the impairment has a substantial and long-term adverse effect on their ability to carry out normal day-to-day activities.

    I believe all of those things you listed were much worse in the 70s (with the potential exception of microplastics) when disability rates were much lower. There is no proof that microplastics are causing autism and ADHD (and a thousand other disabilities). What has changed is diagnostic criteria. In the UK they have become much looser.


  • Why is there never any nuance in these discussions? We can both believe that under-diagnosis occurs, and that over-diagnosis occurs. 20% of all pupils in the UK are now classified as so disabled that they require specialised assistance. “SEND” assistance for this can range from free taxi services to and from school (which recently reached £1.2 billion), to support payments, to special assistants in school. The number of ECHP students (those with the highest needs) increasing by 71%, from 253,679 in 2018 to 434,354 in 2024. SEND spending is out of control.

    So what happened, exactly? The average child disability rate in Europe is 4.6%. How did the UK end up with 20%? Did the UK suffer a catastrophic nuclear event? A war? Famine? None of the above. It is clear that categorisation has become EXTREMELY loose over time on average. This does not mean that there are not children who are struggling to get diagnosed with ADHD. However ADHD and autism are a spectrum disorder. It is not binary. The UK has drawn the line far closer to the normal side of the spectrum than any other nation on Earth. If costs continue to rise at this rate, it risks destabilising the entire health system. Public sentiment will shift, and we risk undermining children getting any diagnosis at all.

    IMHO, this requires at least two tactics at the same time. 1) Invest sufficiently into diagnosis resources. Stringing parents and children along for years while they wait in the system can make the issue much worse than it needs to be. 2) Draw the diagnostic line closer to where the rest of Europe does it. This will mean far fewer children are diagnosed with disabilities, but those who genuinely have a disability are treated much faster and actually receive the resources they need.








  • This is where the data meets an uncomfortable impasse. Hispanics in the U.S. live a lot longer than whites. It’s not because of socio-economics. Hispanics eat a lot of meat, too. It is theorised that their legume heavy diet might be responsible. Meaning that, despite eating a lot of carbs, they still live longer. I suspect longevity effects are primarily related to the fibre. This might be true in the study linked. Perhaps it isn’t the low carb/low fat part which gave heart benefits, but the higher fibre intake.

    As for the mechanisms:

    1. Fibre binds bile acids, which increases bile acid secretion. The liver uses LDL to make more bile acid, reducing LDL.

    2. Fibre acts as a glucose release modulator. Glucose spikes are responsible for a range of immediate and systemic issues. Reducing overall glucose consumption and especially spikes results in lower risk of type 2 diabetes, metabolic syndrome, and atherosclerosis progression.

    3. Lower blood pressure (we’re not really sure why yet).

    4. Fermentable fibres are metabolized by gut bacteria into short-chain fatty acids. These reduce systemic inflammation, improve gut barrier integrity, improve insulin sensitivity, and regulate immune function. The inflammation one in particular could explain a great deal of the health benefits.

    5. The cancer risk specifically decreases because of dilution and faster transit of carcinogens, SCFA production (especially butyrate, which has anti-tumor properties), and reduced insulin and IGF-1 signalling.

    6. Fibre keeps you full for longer, reducing the risk of obesity.